[The place and significance of the autonomic dystonia syndrome in the clinico-pathophysiological structure of the late sequelae of mild closed craniocerebral trauma].

Analysis of the incidence, clinico-pathophysiological structure and dynamics of vegetovascular disorders in subjects with a history of mild closed craniocerebral injuries has demonstrated that in the majority of them, even practically healthy, functional insufficiency of vegetovascular functions is seen for many years after injury. Clinically, it manifests under the influence of diverse harmful exo- and endogenous factors, undergoes circadian changes, is altered during magnetic storms, in the course of traumatic disease and nonmedicamentous correction (by methods of adaptive bioregulation according to heart rhythm parameters, craniocerebral hypothermia, etc). It has been shown that initially transitory, reversible vegetovascular disturbances, provided they were not initially removed, transform with years to more stable vegetotrophic disorders and become risk factor of cerebrovascular diseases in the given group.

[Late sequelae of mild closed cranio-cerebral injuries (incidence, clinical course and criteria of the conditions of compensation and decompensation)].

The authors present the results of clinical catamnestic, clinical neurologic analysis, and of specific neurophysiologic examinations of 292 patients with a history of slight craniocerebral injuries. Late sequelae of these injuries were detected in 70% of the examinees. The criteria and factors of decompensation risk were defined on the basis of the total and clinical structure of the disease, history of the injury, variation pulsometry, bioelectric reactivity, and visual analyzer adaptation function.