Publications by authors named "M Gruhn"

Article Synopsis
  • Childhood adversity (CA) is linked to higher risks of negative health outcomes, with differences in how various types of adversity affect brain structure during development.
  • Recent research indicates that deprivation leads to slower decreases in cortical surface area in certain brain regions, while threat exposure results in increased surface area in areas related to socio-emotional processing as adolescents age.
  • These findings suggest a need to reconsider how different forms of adversity affect brain development over time, highlighting the varying impacts on cognitive and emotional functions.
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It is well-established that disrupted autonomic nervous system (ANS) reactivity exacerbates risk for long-term maladjustment following childhood adversity (CA). However, few studies have integrated measures of both the sympathetic (SNS) and parasympathetic (PNS) branches of the ANS, resulting in a unidimensional understanding of ANS functioning as a mechanism of risk. Further, past work has primarily measured CA only at the aggregate level (e.

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Parental depression symptoms are a prevalent risk factor for internalizing and externalizing problems in youth, with parenting and parents' physiological stress reactivity representing potential contributing factors in the intergenerational transmission of psychopathology symptoms. In a sample of adolescents (N = 97) and their parents, the current study examined parental depression symptoms, an observational measure of parenting, and parents' physiological reactivity during a dyadic conflict discussion task in association with adolescents' internalizing and externalizing psychopathology. Parental depression symptoms and harsh/insensitive parenting showed positive associations with youth psychopathology symptoms.

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Regulatory efforts are hypothesized to affect associations between emotions and physiology (i.e., concordance) to facilitate adaptive functioning.

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Early life adversity (ELA) characterized by threat (e.g., abuse, witnessing violence) impacts neural and physiologic systems involved in emotion reactivity; however, research on how threat exposure impacts the interplay between these systems is limited.

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