Publications by authors named "L T Hannah"
Article Synopsis
- Anchorages are spots where ships wait to go into ports, and more ships have been waiting since 2019 because ports are busy.
- This has raised worries about how the waiting ships might harm the environment and local communities.
- A new tool called the Pathways of Effects Matrix (PoEM) helps show the complicated effects of anchoring and helps communities share their concerns and ideas for solutions.
Article Synopsis
- The Genome in a Bottle Consortium (GIAB) is creating matched tumor-normal samples that are publicly consented for sharing genomic data and cell lines, focusing on pancreatic ductal adenocarcinoma (PDAC).
- They provide a comprehensive genomic dataset from the first individual, combining high-depth DNA from tumor and normal cells using advanced whole genome sequencing technologies.
- This open-access resource aims to help develop benchmarks for detecting genetic variants in cancer, fostering innovation in genome measurement and analysis tools.
The interaction of plant mitochondrial and nuclear genetic systems is exemplified by mitochondria-encoded cytoplasmic male sterility (CMS) under the control of nuclear restorer-of-fertility genes. The S type of CMS in maize is characterized by a pollen collapse phenotype and a unique paradigm for fertility restoration in which numerous nuclear restorer-of-fertility lethal mutations rescue pollen function but condition homozygous-lethal seed phenotypes. Two nonallelic restorer mutations recovered from Mutator transposon-active lines were investigated to determine the mechanisms of pollen fertility restoration and seed lethality.
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- Obesity is linked to insulin resistance, affecting both glucose uptake and the body's blood vessel response to insulin, which is crucial for delivering glucose to tissues.
- A study tested the role of sympathetic activation in impaired insulin-triggered blood vessel dilation in obese adults using a controlled experiment with insulin and drugs to block sympathetic responses.
- Results showed that blocking sympathetic activation significantly improved blood flow and microvascular function when insulin was administered, indicating that sympathetic activity may hinder insulin's vascular effects in obesity.