Thiazolium salt mimics the non-coenzyme effects of vitamin B in rat synaptosomes.

Long-term studies have confirmed a causal relationship between the development of neurodegenerative processes and vitamin B (thiamine) deficiency. However, the biochemical mechanisms underlying the high neurotropic activity of thiamine are not fully understood. At the same time, there is increasing evidence that vitamin B, in addition to its coenzyme functions, may have non-coenzyme activities that are particularly important for neurons.