[The place and significance of the autonomic dystonia syndrome in the clinico-pathophysiological structure of the late sequelae of mild closed craniocerebral trauma].

Analysis of the incidence, clinico-pathophysiological structure and dynamics of vegetovascular disorders in subjects with a history of mild closed craniocerebral injuries has demonstrated that in the majority of them, even practically healthy, functional insufficiency of vegetovascular functions is seen for many years after injury. Clinically, it manifests under the influence of diverse harmful exo- and endogenous factors, undergoes circadian changes, is altered during magnetic storms, in the course of traumatic disease and nonmedicamentous correction (by methods of adaptive bioregulation according to heart rhythm parameters, craniocerebral hypothermia, etc). It has been shown that initially transitory, reversible vegetovascular disturbances, provided they were not initially removed, transform with years to more stable vegetotrophic disorders and become risk factor of cerebrovascular diseases in the given group.