The Treatment of Post-blepharoplasty Lower Eyelid Retraction.

Numerous solutions for post-blepharoplasty lower eyelid retraction are reviewed. Patients require permanent recruitment of skin and soft tissue to lengthen the lower eyelid and control of the lower eyelid shape. The authors use a hand-carved expanded polytetrafluoroethylene (ePTFE) implant held with microscrews to provide volume and felting material at the orbital rim and to permanently fix vertically lifted cheek soft tissue into the lower eyelid.

Revisiting the Safety of Prostaglandin Analog Eyelash Growth Products.

Background: The FDA approved bimatoprost ophthalmic solution 0.03% for treatment of eyelash hypotrichosis in 2008. Consumer concern persists regarding potential side effects of this product.

Pathology Of "Post-Upper Blepharoplasty Syndrome": Implications For Upper Eyelid Reconstruction.

Background: The anatomical basis for eyelid changes after upper blepharoplasty is largely uninvestigated. The post upper blepharoplasty syndrome (PUBS) is here defined as upper eyelid ptosis, hollow sulcus, high or absent upper eyelid crease, eyelash ptosis, loose eyelid platform skin, and compensatory brow elevation.

Objective: The anatomical basis for the post-upper blepharoplasty syndrome was investigated.

Surgical Predictors of Reduced Marginal Reflex Distance After Upper Blepharoplasty.

Purpose: Ptosis may occur in certain cases after upper blepharoplasty. The authors aim to characterize the incidence of postoperative marginal reflex distance (MRD1) reduction after upper blepharoplasty and surgical variables that may predict this occurrence.

Methods: In this cross-sectional study, patients > 18 years old undergoing upper blepharoplasty were screened.

Chlorhexidine Keratitis: Safety of Chlorhexidine as a Facial Antiseptic.

Background: Effective antiseptic to reduce surgical site infections is a cornerstone of modern surgery. Chlorhexidine gluconate-based antiseptics are among the most effective of these products. Unfortunately, chlorhexidine solutions are toxic to the cornea and middle ear, and they pose a splash risk to both the patient and health care personnel.

Persistence of Upper Blepharoptosis After Cosmetic Botulinum Toxin Type A.

Background: Upper eyelid ptosis after cosmetic botulinum toxin is generally considered short-lived and responsive to apraclonidine ophthalmic drops. The authors present a series with persistent ptosis.

Objective: To report a series of patients with persistent upper eyelid ptosis after cosmetic botulinum toxin.

Cosmetic Microdroplet Botulinum Toxin A Forehead Lift: A New Treatment Paradigm.

Purpose: To investigate the safety and efficacy of a microdroplet, cosmetic, periocular botulinum toxin A method that extensively treats the eyebrow depressors but leaves the brow elevators untreated.

Methods: This is a 5-year retrospective, consecutive, nonrandomized series of botulinum toxin treatments. The study was reviewed by an institutional review board and complied with the Health Insurance Portability and Accountability Act (HIPAA).

Traumatic optic neuropathy: an evolving understanding.

Purpose: To critically review the treatment of traumatic optic neuropathy.

Design: A perspective of clinical and basic science studies related to traumatic optic neuropathy and its treatment.

Methods: Published clinical and basic science studies on traumatic optic neuropathy were critically reviewed and interpreted.

Deep-fill hyaluronic acid for the temporary treatment of the naso-jugal groove: a report of 303 consecutive treatments.

Purpose: To report a 2-year experience of treating the naso-jugal groove with injectable hyaluronic acid gel, using a deep-fill method.

Methods: This was a consecutive, retrospective, nonrandomized case series of patients presenting with concerns involving dark circles, lower eyelid hollows, or other contour irregularities that make up the naso-jugal groove. One author performed all treatments, consisting of transcutaneous injection of hyaluronic acid gel filler, to address the naso-jugal groove.

Treatment of traumatic optic neuropathy with high-dose corticosteroid.

Based on the favorable clinical results in acute spinal cord injury, high-dose methylprednisolone at an intravenous loading dose of 30 mg/kg followed by a continuous infusion of 5.4 mg/kg/h for 24 or 48 hours has been adopted for the treatment of acute traumatic optic neuropathy (TON). Although there is anecdotal evidence of the efficacy of high-dose corticosteroid in this condition, there are no prospective, randomized trials to attest to its benefit.

Aesthetic and restorative midface lifting with hand-carved, expanded polytetrafluoroethylene orbital rim implants.

The midface lift represents an important advance in aesthetic and reconstructive surgery. However, the need for reliable fixation along the orbital rim has been a significant challenge. Furthermore, volume is needed at the orbital rim, to compensate for long-term remodeling of the bone of the orbital rim and malar face.

Methylprednisolone exacerbates axonal loss following optic nerve trauma in rats.

Purpose: This study investigates the clinical dogma that very high doses of methylprodnisolone helpful in spinal cord injury are also helpful in optic nerve trauma. Methods: The right optic nerve of 29 male rats received a 5 second traumatic crush followed 30 minutes later by one of five intravenous treatments (methylprednisolone 30 mg/kg, 60 mg/kg, 90 mg/kg, 120 mg/kg, or saline). Treatment was continued for three additional administrations at 6 hour intervals.