Anemia management in peritoneal dialysis patients: can an iron supplement maintain a normal transferrin saturation and hemoglobin level?

The primary cause of anemia in dialysis patients is inadequate production of erythropoietin (EPO) by the dysfunctional kidneys. The EPO circulates in plasma and acts on erythroid progenitor cells in the bone marrow to produce red blood cells (RBCs). At the same time, chronic inflammatory diseases reduce the release of iron from storage sites, resulting in low transferrin saturation (Fe+ sat%).

Potassium management in pediatric peritoneal dialysis patients: can a diet with increased potassium maintain a normal serum potassium without a potassium supplement?

Hypokalemia can result from an absence of cellular redistribution accompanied by low serum potassium levels, which can be secondary to inadequate dietary intake, external losses (for example, through the gut or skin), or renal losses. However, low serum potassium secondary to low potassium ingestion usually occurs after an extended period of low oral potassium intake. Also, increased renal potassium excretion can be the result of magnesium deficiency.