Publications by authors named "Kaisheng Jiang"

Morphology of right ventricular outflow tract (RVOT) is potentially related to cardiovascular outcomes. However, this relationship still remains to be verified with direct evidence. We retrospectively reviewed cases from the autopsy specimen library in the Center of Forensic Medicine in Sun Yat-sen University from 2017 to 2023.

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Hydrogen sulfide (HS) detection remains a significant concern and the sensitivity, selectivity, and detection limit must be balanced at low temperatures. Herein, we utilized a facile solvothermal method to prepare Cu-doped SnO/rGO nanocomposites that have emerged as promising candidate materials for HS sensors. Characterization of the Cu-SnO/rGO was carried out to determine its surface morphology, chemical composition, and crystal defects.

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HS is a poisonous gas that is widespread in nature and human activities. Its rapid and sensitive detection is essential to prevent it from damaging health. Herein, we report Pd- and Pt-bimetallic-nanoparticle-doped InO hollow microspheres that are synthesized using solvothermal and in situ reduction methods for HS detection.

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In this work, SnO nanoflowers were prepared by a simple one-step hydrothermal process. The morphology and structure of SnO nanoflowers were characterized by SEM, TEM, Raman spectroscopy, and XRD, which demonstrated the good crystallinity of the SnO tetrahedron structure of the as-synthesized materials. In addition, the sensing properties of SnO nanoflowers were studied in detail.

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Hydrogen (H) sensors are of great significance in hydrogen energy development and hydrogen safety monitoring. However, achieving fast and effective detection of low concentrations of hydrogen is a key problem to be solved in hydrogen sensing. In this work, we combined the excellent gas sensing properties of tin(IV) oxide (SnO) and zinc oxide (ZnO) with the outstanding electrical properties of reduced graphene oxide (rGO) and prepared palladium (Pd)-doped rGO/ZnO-SnO nanocomposites by a hydrothermal method.

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Application of chemotherapeutic oxaliplatin represses gene transcription through induction of DNA methylation, which may contribute to oxaliplatin-induced chronic pain. Here, Ddr1, which showed an increased methylation in the promoter, was screened from the SRA methylation database (PRJNA587622) after oxaliplatin treatment. qPCR and MeDIP assays verified that oxaliplatin treatment increased the methylation in Ddr1 promoter region and decreased the expression of DDR1 in the neurons of spinal dorsal horn.

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