LCAT converts free cholesterol to cholesteryl esters in the process of reverse cholesterol transport. Familial LCAT deficiency (FLD) is a genetic disease that was first described by Kaare R. Norum and Egil Gjone in 1967.
View Article and Find Full Text PDFWe translated two Latin texts about scurvy. One is by Ambrosius Rhodius, who in 1635 published his doctoral thesis on scurvy. This contains aspects of 16th- and 17th-century folklore medicine.
View Article and Find Full Text PDFBackground: In 2006, the World Health Organization (WHO) launched the new global WHO Child Growth Standards for children under five years. The rationale for developing this new growth standard was that a review of the NCHS/WHO-reference, which had been recommended for international use since 1978, showed that the growth of breast-fed infants deviated negatively from this reference. Most children included in the NCHS/WHO-reference from 1978 were fed mainly with infant formula.
View Article and Find Full Text PDFThe World Health Organization (WHO) Multicentre Growth Reference Study (MRGS) European site was Oslo, Norway. Oslo has a high breastfeeding rate. Ninety-nine percent of mothers initiate breastfeeding soon after delivery, and 80% continue for at least six months.
View Article and Find Full Text PDFWe examined the liver of adult polar bears, arctic foxes, and rats by gold chloride staining, fluorescence microscopy for the detection of autofluorescence of vitamin A, hematoxylin-eosin staining, staining with Masson's trichrome, Ishii and Ishii's silver impregnation, and transmission electron microscopical morphometry. The liver lobules of the arctic animals showed a zonal gradient in the storage of vitamin A. The density (i.
View Article and Find Full Text PDFTidsskr Nor Laegeforen
June 2002
Axel Holst (1860-1931), professor of hygiene and bacteriology at the University of Oslo and paediatrician Theodor Frølich (1870-1947) became interested in a disease termed "ship beriberi" which afflicted the crews of sailing ships, and which showed an uncanny likeness to scurvy. They suspected a nutritional deficiency, and established an animal model that allowed systematic study of factors that led to disease as well as the preventive value of different substances. The choice of the guinea pig as the experimental animal for these studies was one indeed fortuitous, as that species has been shown to be among the very few mammals incapable of endogenous synthesis of ascorbic acid.
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