Lycopene: an antioxidant product reducing dithane toxicity in Allium cepa L.

The current study was undertaken to assess the attenuating potential of lycopene against Dithane toxicity in Allium cepa L. roots. A.

Turmeric (Curcuma longa L.) tends to reduce the toxic effects of nickel (II) chloride in Allium cepa L. roots.

The immense protection potential of plant-derived products against heavy metal toxicity has become a considerable field of research. The goal of the present study was to evaluate the mitigative ability of turmeric against nickel (II) chloride (NiCl)-related toxicity in the roots of Allium cepa L. For this purpose, one control (treated with tap water) and five treatment groups (treated with 440 mg/L turmeric, 880 mg/L turmeric, 1 mg/L NiCI, 1 mg/L NiCI + 440 mg/L turmeric, and 1 mg/L NiCI + 880 mg/L turmeric, respectively) of Allium bulbs were established.

Molecular docking and toxicity assessment of spirodiclofen: protective role of lycopene.

In this study, toxic effects of spirodiclofen and protective role of lycopene against toxic effects were investigated by using physiological, cytogenetic, anatomical, and biochemical parameters. Allium cepa L. bulbs were used as test material.

Hydrogen sulfide dilates the isolated retinal artery mainly via the activation of myosin phosphatase.

Aims: Hydrogen sulfide (HS) is shown in ocular tissues and suggested to involve in the regulation of retinal circulation. However, the mechanism of HS-induced relaxation on retinal artery is not clarified yet. Herein, we aimed to evaluate the role of several calcium (Ca) signaling and Ca sensitization mechanisms in the relaxing effect of HS donor, NaHS, on retinal arteries.

In vivo protective effects of Ginkgo biloba L. leaf extract against hydrogen peroxide toxicity: cytogenetic and biochemical evaluation.

In this study, the protective effects of Ginkgo biloba leaf extract (GbE) against toxicity induced by hydrogen peroxide (HO) in Swiss albino mice were investigated. Abnormal metaphase number (AMn), mitotic index (MI), micronucleus (MN), and chromosomal abnormalities (CAs) were analyzed for cytogenetic effects. Serum aspartate aminotransferase (AST), alanine aminotransferase (ALT), alkaline phosphatase (ALP), blood urea nitrogen (BUN), creatinine, glutathione (GSH), and malondialdehyde (MDA) levels in liver and kidney organs were investigated as indicators of biochemical toxicity.