Publications by authors named "K Y Larbi"

Background: The chemical composition and biological activities of Eucalyptus essential oils (EOs) have been documented in numerous studies against multiple infectious diseases. The antibacterial activity of individual Eucalyptus EOs against strains that cause ear infections was investigated in our previous study. The study's antibacterial activity was promising, which prompted us to explore this activity further with EO blends.

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Objectives: Daily calcium supplements are recommended for pregnant women from 20 weeks' gestation to prevent pre-eclampsia in populations with low dietary calcium intake. We aimed to improve understanding of barriers and facilitators for calcium supplement intake during pregnancy to prevent pre-eclampsia.

Design: Mixed-method systematic review, with confidence assessed using the Grading of Recommendations, Assessment, Development and Evaluations-Confidence in the Evidence from Reviews of Qualitative research approach.

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Background: Hypertension is responsible for 8.5 million deaths from stroke, ischemic heart disease and other vascular and kidney diseases. In Algeria, hypertension (HTN) is the most common chronic disease with an estimated prevalence of 23,6%.

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Introduction: Low dietary calcium intake is a risk factor for pre-eclampsia, a major contributor to maternal and perinatal mortality and morbidity worldwide. Calcium supplementation can prevent pre-eclampsia in women with low dietary calcium. However, the optimal dose and timing of calcium supplementation are not known.

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Article Synopsis
  • - Autosomal dominant polycystic kidney disease (ADPKD) is a genetic condition causing multiple cysts in kidneys, primarily due to mutations in the PKD1 and PKD2 genes that code for polycystin proteins.
  • - Researchers analyzed DNA from 90 kidney cysts from 24 patients, discovering that 93% of these cysts had harmful somatic mutations in PKD1 or PKD2, mainly resulting in significant gene alterations like truncations.
  • - The study suggests that cyst formation in ADPKD follows a cellular recessive mechanism, implicating both inherited and acquired mutations in the PKD1 and PKD2 genes within kidney cyst cells.
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