Publications by authors named "K Takai"
Angiocentric glioma (AG) is a supratentorial diffuse low-grade glioma characterized by the MYB::QKI fusion gene, showing angiocentric growth of monomorphous spindle cells with astrocytic and ependymal immunophenotypes. We describe a rare case of MYB::QKI fusion-positive diffuse cerebellar glioma in a 54-year-old male. The patient initially presented with a T2/FLAIR hyperintense lesion in the left cerebellar hemisphere and slowly progressive neurological symptoms.
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- - A 59-year-old woman developed complex regional pain syndrome in her right arm after surgery for cubital tunnel syndrome, leading to treatment with spinal cord stimulation (SCS) that initially helped her pain.
- - Four years later, she experienced motor weakness on her left side and reduced effectiveness of the SCS therapy, which prompted an MRI revealing significant compression of her cervical spinal cord due to epidural granulation tissue.
- - After surgically removing the granulation tissue, her motor weakness improved, and histological tests indicated that an allergic reaction to materials in the SCS setup caused the tissue fibrosis.
A novel mesophilic bacterium, strain SS33, was isolated from a deep-sea hydrothermal vent chimney at Suiyo Seamount, Izu-Bonin Arc, Western Pacific Ocean. The cells of strain SS33 were motile short rods with a single polar flagellum. The growth of strain SS33 was observed at the temperature range between 33 and 55 °C (optimum growth at 45 °C), at the pH range between 5.
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- Hepatocellular carcinoma (HCC) rupture can lead to severe complications, including a rare condition called non-occlusive mesenteric ischemia (NOMI), which has been underreported.
- A 77-year-old man with an HCC experienced abdominal pain and shock; after treatment to stop bleeding, his condition improved temporarily but worsened again, leading to further diagnostic imaging.
- The patient required surgery for affected intestines and was diagnosed with NOMI, marking the first documented case of this complication following HCC rupture, highlighting the need for awareness and prompt treatment of such cases.
Objective: Patients with spinal CSF leaks often have ventral dural abnormalities (type 1 CSF leaks); however, the pathological mechanism for developing dural abnormalities is unknown. The authors investigated whether calcified dural ligaments contribute to the development of ventral dural tears, which cause spinal CSF leaks.
Methods: Consecutive patients diagnosed with type 1 CSF leaks who had spiculated spinal lesions between 2010 and 2024 were included.