Nutritional therapy for reducing disability and improving activities of daily living in people after stroke.

Background: Stroke patients often face disabilities that significantly interfere with their daily lives. Poor nutritional status is a common issue amongst these patients, and malnutrition can severely impact their functional recovery post-stroke. Therefore, nutritional therapy is crucial in managing stroke outcomes.

Contrast-Enhanced Ultrasonography in Diagnosing Intravascular Large B-Cell Lymphoma Infiltrating Liver Sinusoids.

BACKGROUND Intravascular large B-cell lymphoma (IVLBCL) is a rare extranodal large B-cell lymphoma characterized by the selective growth of lymphoma cells within vasculature. This presents a diagnostic challenge due to non-specific symptoms and lack of tumor formation. 18F-fluorodeoxyglucose positron emission tomography/computed tomography (FDG PET/CT) provides useful information in diagnosing FDG-avid lymphoma, but is not specific to  IVLBCL.

Direct Detection of Long-Range Interdomain Auger Recombination in Dumbbell-Shaped Quasi-Type-II Nanoparticle.

Multicarrier dynamics in heterostructured ZnS-AgInS (ZAIS) dumbbell-like nanoparticle (nanodumbell), which consists of two visible-light absorptive domains (ellipsoidal tip domains) directly linked to each end of a 22 nm length rod domain of the ZAIS nanodumbell with a quasi-type-II heterostructure, was investigated by femtosecond transient absorption spectroscopy under variable excitation intensities. Quantitative analysis together with the numerical simulations for the excitation intensity dependence of the dynamics revealed that only one electron-hole pair survived in the overall dumbbell as a consequence of Auger recombination, even though multiple carriers were formed on both terminal tip domains. This result strongly suggested carrier-carrier interaction between the tip domains, leading to the long-range Auger recombination via tunneling across a rod potential barrier.

Mild hypothermia ameliorates anesthesia toxicity in the neonatal macaque brain.

Sedatives and anesthetics can injure the developing brain. They cause apoptosis of neurons and oligodendrocytes, impair synaptic plasticity, inhibit neurogenesis and trigger long-term neurocognitive deficits. The projected vulnerable period in humans extends from the third trimester of pregnancy to the third year of life.

Quantitative ultrasound and apoptotic death in the neonatal primate brain.

Apoptosis is triggered in the developing mammalian brain by sedative, anesthetic or antiepileptic drugs during late gestation and early life. Whether human children are vulnerable to this toxicity mechanism remains unknown, as there are no imaging techniques to capture it. Apoptosis is characterized by distinct structural features, which affect the way damaged tissue scatters ultrasound compared to healthy tissue.