Power-Law Entanglement and Hilbert Space Fragmentation in Nonreciprocal Quantum Circuits.

Quantum circuits utilizing measurement to evolve a quantum wave function offer a new and rich playground to engineer unconventional entanglement dynamics. Here, we introduce a hybrid, nonreciprocal setup featuring a quantum circuit, whose updates are conditioned on the state of a classical dynamical agent. In our example the circuit is represented by a Majorana quantum chain controlled by a classical N-state Potts chain undergoing pair flips.

Eosinophils Suppress the Migration of T Cells Into the Brain of -Infected Mice and Protect Them From Experimental Cerebral Malaria.

Cerebral malaria is a potentially lethal disease, which is caused by excessive inflammatory responses to parasites. Here we use a newly developed transgenic ANKA () parasite that can be used to study parasite-specific T cell responses. Our present study demonstrates that mice, which lack type I interferon receptor-dependent signaling, are protected from experimental cerebral malaria (ECM) when infected with this novel parasite.

Time-Domain Anyon Interferometry in Kitaev Honeycomb Spin Liquids and Beyond.

Motivated by recent experiments on the Kitaev honeycomb magnet α-RuCl_{3}, we introduce time-domain probes of the edge and quasiparticle content of non-Abelian spin liquids. Our scheme exploits ancillary quantum spins that communicate via time-dependent tunneling of energy into and out of the spin liquid's chiral Majorana edge state. We show that the ancillary-spin dynamics reveals the edge-state velocity and, in suitable geometries, detects individual non-Abelian anyons and emergent fermions via a time-domain counterpart of quantum-Hall anyon interferometry.

Hydrodynamic Stabilization of Self-Organized Criticality in a Driven Rydberg Gas.

Signatures of self-organized criticality (SOC) have recently been observed in an ultracold atomic gas under continuous laser excitation to strongly interacting Rydberg states [S. Helmrich et al., Nature, 577, 481-486 (2020)].

Endophilin A2 deficiency protects rodents from autoimmune arthritis by modulating T cell activation.

The introduction of the CTLA-4 recombinant fusion protein has demonstrated therapeutic effects by selectively modulating T-cell activation in rheumatoid arthritis. Here we show, using a forward genetic approach, that a mutation in the SH3gl1 gene encoding the endocytic protein Endophilin A2 is associated with the development of arthritis in rodents. Defective expression of SH3gl1 affects T cell effector functions and alters the activation threshold of autoreactive T cells, thereby leading to complete protection from chronic autoimmune inflammatory disease in both mice and rats.