Publications by authors named "K J Bianchini"

Cadmium (Cd) is a trace element of toxicological concern that has been monitored in marine birds inhabiting the Canadian Arctic since 1975. Despite nearly 50 years of monitoring, research to date has largely evaluated single species, locations, or time points, and there is as of yet no holistic overview that jointly considers all available Cd data. We addressed this information gap by combining and analyzing most of the existing data on hepatic Cd concentrations in marine birds from the Canadian Arctic.

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Polycyclic aromatic hydrocarbons (PAHs) impaired pre-migratory fueling in 49 orally dosed Sanderling (Calidris alba). In the present study, 8 genes related to fat deposition and PAH exposure were measured in liver subsamples from these same shorebirds. At the highest dose (1260 µg total PAH [tPAH]/kg body wt/day), PAH exposure decreased liver basic fatty acid binding protein 1 (Lbfabp) and hepatic lipase (Lipc) expression.

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Differences in avian sensitivity to dioxin-like compounds (DLCs) are directly attributable to the identities of amino acids at two sites within the ligand binding domain (LBD) of the aryl hydrocarbon receptor 1 (AHR1). Recent work suggests that by influencing avian exposure to naturally occurring dioxins, differences in diet, habitat, and migration may have influenced the evolution of three AHR1 LBD genotypes in birds: type 1 (high sensitivity), type 2 (moderate sensitivity), and type 3 (low sensitivity). Using a boosted regression tree (BRT) analysis, we built on previous work by examining the relationship between a comprehensive set of 17 species traits, phylogeny, and the AHR1 LBD across 89 avian species.

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Common loons (Gavia immer) are top predators that depend on lake food webs to successfully fledge chicks. Common loon reproductive success is consequently recognized as an important indicator of aquatic ecosystem health. Existing evidence points to long-term declines in productivity in portions of the common loon range; however, the reason for these declines is not well understood.

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Intentional exaggeration of symptoms is a potential problem in contexts where there are financial incentives to appear disabled. Therefore, calibration of tools to accurately evaluate malingering in these contexts is important. The present study used a criterion groups validation design to determine the ability of the Pain Catastrophizing Scale (PCS) to detect Malingered Pain-Related Disability (MPRD).

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