[Right ventricular remodeling after myocardial infarctions of different location].

Aim: To study dynamic dimensional and functional changes of both ventricles after myocardial infarctions (MI) of different locations.

Material And Methods: Patients with first acute ST elevation MI (n=180) were distribution to 3 groups: anterior (AMI), inferior (IMI), and posterolateral (PLMI). EchoCG dimensional parameters of LV and RV as well as functional two dimensional EchoCG, flow and tissue doppler parameters were measured on days 7, 90 and after 1 year of follow up to reveal systolic or diastolic dysfunction of both chambers.

[Study of morphofunctional characteristics of the myocardium in ischemic heart disease using ultrasonic methods].

Ultrasonic heart tissue characterization was performed in normal subjects and patients with chronic coronary disease and acute myocardial infarction (MI). In normal subjects and anginal patients, no regional differences in myocardial echo density were found, while postinfarction patients showed an increase of echo density in the region of the scar. During MI, initial fall of echo density in the damaged area at the onset of the disease was followed by an increase of echo density later in the course of MI.

[Correlations between various cardiometric and functional parameters in dilated cardiomyopathy and alcoholic lesion of the heart (data of roentgenological and echocardiographic studies)].

Volumetric changes of the heart and its left ventricle, as well as myocardial contractility were assessed in 50 patients with dilatation cardiomyopathy (DCMP) and 16 patients with acute alcoholic heart damage (AHD), on the basis of roentgenologic (cardiac shape and volume, left ventricular and aortic pulsation, respiratory volumetric variations, pulmonary circulation) and echocardiographic (end-diastolic size and volume, systolic increment of posterior wall thickness, ejection fraction and left-ventricular shortening) data. Both DCMP and acute AHD were shown to be associated with total enlargement of the heart, diffuse impairment of myocardial contractility, considerably depressed left ventricular pumping function, early congestion in the systemic circulation network coupled with minor roentgenologic signs of congestion in pulmonary circulation.