Three-Dimensional Graphene Sheet-Carbon Veil Thermoelectric Composite with Microinterfaces for Energy Applications.

Over the years, various processing techniques have been explored to synthesize three-dimensional graphene (3DG) composites with tunable properties for advanced applications. In this work, we have demonstrated a new procedure to join a 3D graphene sheet (3DGS) synthesized by chemical vapor deposition (CVD) with a commercially available carbon veil (CV) via cold rolling to create 3DGS-CV composites. Characterization techniques such as scanning electron microscopy (SEM), Raman mapping, X-ray diffraction (XRD), electrical resistance, tensile strength, and Seebeck coefficient measurements were performed to understand various properties of the 3DGS-CV composite.

The Factors Associated with the Development of COVID-19 Symptoms among Employees in a U.S. Healthcare Institution.

Healthcare workers have experienced increased occupational health risks caused by COVID-19 disease. The purpose of this project was to examine the relationships between reporting COVID-19 symptoms by employees in a healthcare institution in the United States and employees' demographics, vaccination status, co-morbid conditions, and body mass index (BMI). This project employed a cross-sectional design.

Deficiency of fatty acid-binding proteins in mice confers protection from development of experimental autoimmune encephalomyelitis.

Fatty acid-binding proteins (FABPs) act as intracellular receptors for a variety of hydrophobic compounds, enabling their diffusion within the cytoplasmic compartment. Recent studies have demonstrated the ability of FABPs to simultaneously regulate metabolic and inflammatory pathways. We investigated the role of adipocyte FABP and epithelial FABP in the development of experimental autoimmune encephalomyelitis to test the hypothesis that these FABPs impact adaptive immune responses and contribute to the pathogenesis of autoimmune disease.

Chemical genetic screening identifies critical pathways in anthrax lethal toxin-induced pathogenesis.

Anthrax lethal toxin (LT)-induced cell death via mitogen-activated protein kinase kinase (MAPKK) cleavage remains questionable. Here, a chemical genetics approach was used to investigate what pathways mediate LT-induced cell death. Several small molecules were found to protect macrophages from anthrax LT cytotoxicity and MAPKK from cleavage by lethal factor (LF), without inhibiting LF enzymatic activity or cellular proteasome activity.