Motion sickness history, food neophobia, and sensation seeking.

Motion sickness is believed to be caused by conflicting sensory signals, a situation that mimics the effects of ingesting certain toxins. Thus, one might suspect that individuals who have experienced a relatively high frequency of motion sickness may be particularly vigilant about avoiding anything that produces nausea, induding potentially nauseating toxins. Consequently, they may be more resistant to trying new foods, i.

Impact of postnatal glucocorticoids on early lung development.

Inhaled glucocorticoid treatment during the first 2 yr of life is controversial because this is a period of major structural remodeling of the lung. Rabbits received aerosolized budesonide (Bud; 250 microg/ml) or injected dexamethasone (Dex; 0.05 mg.

Variability in preterm lamb lung mechanics after intra-amniotic endotoxin is associated with changes in surfactant pool size and morphometry.

Antenatal exposure to intra-amniotic (i.a.) endotoxin initiates a complex series of events, including an inflammatory cascade, increased surfactant production, and alterations to lung structure.

Protective Effect of EGb 761 Against Oxidative Phosphorylation of Brain Mitochondria After Anoxia/Reoxygenation In Vivo and In Vitro.

The aim of this study was to investigate the effects of in vivo and in vitro anoxia and reoxygenation on the oxidative phosphorylation of brain mitochondria and to study the protective effects of Ginkgo biloba extract (EGb 761). Cerebral ischemia and reperfusion induced slight decreases in respiration rates and in the efficiency of oxidative phosphorylation. Total protection of mitochondrial function was observed after chronic pretreatment of rat with EGb 761.