Single-Center Experience of Surgical Treatment of Primary Retroperitoneal Tumors.

This study is an evaluation of surgical treatment results of primary retroperitoneal tumors. In Samara Regional Clinical Oncology Dispensary, from 2008 to 2015, the treatment of 187 patients (53 men and 134 women) was conducted. One hundred fifteen patients got tumor removal within the healthy tissue (R0), and 61 patients went through complete resection of tumor with wide margins (R0).

[Ultrastructure of gingival blood capillary endotheliocytes in patients with chronic catarrhal gingivitis in the presence of connective tissue dysplasia].

Endotheliocyte structure in the gingival capillaries of patients with chronic catarrhal gingivitis (CCG) in the presence of connective tissue dysplasia and arterial hypertension differs from that in patients with CCG without somatic diseases, which essentially increase the permeability of capillary walls.

[The ultrastructure of gingival epithelial prickle layer cells in patients with chronic catarrhal gingivitis concomitant with connective tissue dysplasia].

Patients with chronic gingivitis and connective tissue dysplasia developed more marked dystrophic process in the gingival epithelium than those with chronic gingivitis without general somatic diseases. These dystrophic processes substantially increase the barrier properties of the gingival epithelium.

[A morphological assessment of the efficacy of the deaggregation therapy of chronic catarrhal gingivitis].

Light optics, electron microscopy, morphometry were used to study the structural changes in the epithelial cells and endotheliocytes of gingival papillary capillaries of patients with chronic catarrhal gingivitis after papaverin therapy. The studies have revealed signs of disappearance of intercellular edema and a higher degree of cellular differentiation; signs of normalization of transcellular fluid transport were seen in the endotheliocytes, and aggregations of formed elements of the blood disappeared.

[Pathogenesis of phantom limb syndrome and its treatment].

The spinal mechanism of the phantom pain origin is proved. The underlying factors are: segmentary spinal denervation hypersensitivity and attenuation of the descending inhibitory influence. The pathogenetic treatment of phantom pains is proposed by means of an associated treatment by GABA-ergic drug--Baclofen and alpha-2--adrenomimetic--Clofelin.