Facet-Dependent Cuprous Oxide Nanocrystals Decorated with Graphene as Durable Photocatalysts under Visible Light.

Three morphologies (octahedral, hierarchical and rhombic dodecahedral) of crystal Cu₂O with different facets ({111}, {111}/{110}, and {110}) incorporating graphene sheets (denoted as o-Cu₂O-G, h-Cu₂O-G and r-Cu₂O-G, respectively) have been fabricated by using simple solution-phase techniques. Among these photocatalysts, the r-Cu₂O-G possesses the best photocatalytic performance of 98% removal efficiency of methyl orange (MO) with outstanding kinetics for 120 min of visible light irradiation. This enhancement is mainly due to the dangling “Cu” atoms in the highly active {110} facets, resulting in the increased adsorption of negatively charged MO.

Highly visible-light-responsive CuO/rGO decorated with FeO@SiO nanoparticles as a magnetically recyclable photocatalyst.

The rhombic dodecahedral cuprous oxide-reduced graphene oxide/core-shell FeO@SiO composites (denoted as rCuO-rGO/FeO@SiO) are successfully synthesized facilely via a wet-chemical route. The resulting rCuO-rGO/FeO@SiO combines the unique structure of CuO, electronic characteristics of reduced graphene oxide (rGO) and magnetic property of FeO@SiO to be an effective and recoverable photocatalyst for the degradation of methyl orange (MO). The obtained results show that rCuO-rGO/FeO@SiO is capable of completely degrading MO in the presence of a very low catalyst concentration (0.

Visible-light-driven photodegradation of sulfamethoxazole and methylene blue by Cu2O/rGO photocatalysts.

The cuprous oxide-reduced graphene oxide (Cu2O/rGO-x) composites were prepared via a simple wet-chemical method by using CuSO4·5H2O and graphene oxide as precursors and ascorbic acid as a reducing agent, respectively. These Cu2O/rGO-x were employed as photocatalysts for degrading emerging contaminants and organic dye pollutants (i.e.

[Effect of PP60c-Src on angiotensin II-induced signal transduction in rat vascular smooth muscle cells].

Objective: To further clarify the mechanism of Ang II-induced intracellular signal transduction in vascular smooth muscle cells(VSMCs) proliferation by observing the effect of c-Src on Ang II-mediated MAPK activation and c-fos protein expressions in rat VSMCs.

Methods: Aortic VSMCs from SD rats were cultured primarily and subcultured, which were transfected with anti-sense c-Src oligodeoxynucleotides(ODNs) wrapped with lipofectin to inhibit c-Src activity and protein production. Untransfected VSMCs were used as control, to observe the role of Ang II stimulation in MAPK activation and c-fos protein expression in VSMC.