[Lipoprotein (a), D-Dimer and apolipoprotein A1 as markers of presence and severity of coronary disease].

Background And Objective: To examine the relationship between blood levels of lipids as well as haemostatic and inflammatory markers and the presence and severity of angiographically-evaluated coronary stenosis.

Material And Method: We included 897 patients (629 males and 268 females), aged 65.12+/-11.

Acute versus chronic myocardial ischemia: a differential biological profile study.

Objective: To determine the possible differences in lipid, thrombogenic and inflammatory marker concentrations and the presence of chronic and acute coronary artery disease (stable and unstable angina, respectively), comparing them with a group of control patients with normal coronary arteries.

Material And Methods: This prospective cohort study included 125 patients with unstable angina, 189 with stable angina and a control group of 83 patients with normal coronary arteries. Marker concentrations were measured in all 3 groups.

[Predictive value of lipoprotein (a) and apolipoprotein A1 in patients with angiographycally evaluated coronary stenosis].

Background And Objective: To examine the relationship between blood levels of lipids, hemostatic and inflammatory markers and the presence of angiographycally evaluated coronary stenosis.

Patients And Method: We included 397 consecutive patients (267 males and 130 females) who were admitted to the hospital because of an acute episode of chest pain. Each patient underwent a coronariography using the Seldinger technique.

Prevalence and biologic profile of aspirin resistance in patients with angiographically proven coronary artery disease.

Background: Aspirin protects from cardiovascular events. However, a number of patients who take this drug suffer events, probably due to aspirin resistance. The role of certain biologic variables that may affect resistance is still uncertain.

Association between inflammation, lipid and hemostatic factors in patients with stable angina.

Introduction: Dyslipidemia and thrombotic processes are both clearly involved in atherogenesis and its secondary complications. Moreover, inflammation has also been shown to play an important role in the pathophysiology of atherosclerosis. Our objective was to determine the association between inflammation, lipids and thrombosis in a group of patients with stable angina.