Cross Sectional Study among Intraocular Pressure, Mean Arterial Blood Pressure, and Serum Testosterone according to the Anthropometric Obesity Indices in Korean Men.

Purpose: To investigate the association between intraocular pressure (IOP) and serum testosterone according to the severity of obesity.

Materials And Methods: We investigated the correlation of IOP-testosterone according to the anthropometric obesity indices, such as obesity index (OI), body mass index (BMI), and waist circumference to height ratio (WtHR). Each of the obesity indices was classified into three groups: normal, overweight, and obesity.

Effects of repetitive photodynamic therapy using indocyanine green for acne vulgaris.

Background: Indocyanine green (ICG) is a photosensitizer recently introduced for the treatment of acne.

Objective: To evaluate the efficacy and safety of photodynamic therapy (PDT) using ICG in subjects with acne vulgaris and to evaluate whether there was a difference in the efficacy of ICG-PDT between different numbers of treatment.

Materials And Methods: Subjects with acne on the face were included.

Intraocular toxicity and pharmacokinetics of candesartan in a rabbit model.

Purpose: To investigate the intravitreal toxicity and pharmacokinetics of candesartan, a selective type 1 angiotensin II receptor blocker, in rabbit eyes.

Methods: For the toxicity study, 15 white rabbits were divided into three groups (five rabbits each). Different candesartan doses, namely 0.

The effect of the YK lens in keratoconus.

Purpose: To report the clinical results of the use of the YK contact lens (Lucid Co., Seoul, Korea) for keratoconus.

Methods: One hundred and twenty nine keratoconic eyes were fitted with a YK lens which has one base curve and three peripheral curves.

Identification of a novel mutation in the EYA1 gene in a Korean family with branchio-oto-renal (BOR) syndrome.

The branchio-oto-renal (BOR) syndrome is an autosomal dominant disorder characterized by the association of branchial cysts or fistulae, external ear malformation and/or preauricular pits, hearing loss, and renal anomalies. Mutations in the EYA1 gene on the chromosome band 8q13.3, the human homologue of the Drosophila eyes absent (eya) gene, have been identified to be the underlying genetic defects of the syndrome.