Proteomic clues to the identification of QX disease-resistance biomarkers in selectively bred Sydney rock oysters, Saccostrea glomerata.

The Sydney rock oyster, Saccostrea glomerata, is susceptible to infection by the protozoan parasite, Marteilia sydneyi, the causative agent of QX disease. M. sydneyi infection peaks during summer when QX disease can cause up to 95% mortality.

Breeding for QX disease resistance negatively selects one form of the defensive enzyme, phenoloxidase, in Sydney rock oysters.

QX disease in Sydney rock oysters (Saccostrea glomerata) is caused by the paramyxean protozoan, Marteilia sydneyi. Disease outbreaks occur during summer (January to May) and can result in up to 95% mortality. The New South Wales Department of Primary Industries has been selectively breeding S.

The use of the oyster Saccostrea glomerata as a biomonitor of trace metal contamination: intra-sample, local scale and temporal variability and its implications for biomonitoring.

Cu, Cd, Zn, Pb and Se concentrations were measured in the bivalve mollusc Saccostrea glomerata(Iredale and Roughly) from two uncontaminated locations, Clyde River Estuary, Batemans Bay and Moona Moona Creek, Jervis Bay, to determine natural variability of metals associated with mass, gender, age, tissue type and site within location. Trace metals were also measured in the Clyde River Estuary over an 11 year period and in five other NSW estuaries (Hastings River, Hunter River, Georges River, Tillgerry Creek and Lake Pambula) over a 13-month period to determine temporal variability and if diploid and triploid oysters accumulate trace metals differently. There were few significant relationships between trace metal concentrations and mass and no significant differences in trace metal concentrations between female and male oysters.