Interactions among heavy metals and methane-metabolizing microorganisms and their effects on methane emissions in Dajiuhu peatland.

Peatlands play a crucial role in mediating the emissions of methane through active biogeochemical cycling of accumulated carbon driven by methane-metabolizing microorganisms; meanwhile, they serve as vital archives of atmospheric heavy metal deposition. Despite many edaphic factors confirmed as determinants to modulate the structure of methanotrophic and methanogenic communities, recognition of interactions among them is limited. In this study, peat soils were collected from Dajiuhu peatland to assess the presence of heavy metals, and methanotrophs and methanogens were investigated via high-throughput sequencing for functional genes mcrA and pmoA.

[MIL-88A@MIP Activated Persulfate for Targeted Degradation of Dibutyl Phthalate].

MIL-88A@MIP was fabricated for the first time in this experiment with a metal-organic framework of MIL-88A as the precursor based on the molecular imprinting method. It was characterized by X-ray diffraction (XRD), scanning electronic microscopy (SEM), energy dispersive spectrometer (EDS), and N adsorption. The catalytic performance of MIL-88A@MIP was tested to activate persulfate (PS) to generate SO for the degradation of dibutyl phthalate (DBP), which was used as a target pollutant.

Activation performance and mechanism of a novel heterogeneous persulfate catalyst: Metal Organic Framework MIL-53(Fe) with Fe/Fe mixed-valence coordinative unsaturated iron center.

In this work, a novel effective heterogeneous catalyst metal-organic framework MIL-53(Fe) has been synthesized for the purpose of activating persulfate (PS). Catalytic performance of MIL-53(Fe) activated under different vacuum conditions was investigated; stability and reusability of the catalyst were evaluated, and the activation mechanism was also investigated. The results indicated that vacuum activation could cause variation of the Fe/Fe relative amount ratio of the catalyst, and thus would change the catalytic activities of MIL-53(Fe), because Fe or Fe CUS (coordinative unsaturated metal site) are alternative active sites.

Enhancement of death receptor 4-mediated apoptosis and cytotoxicity in renal cell carcinoma cells by anisomycin.

Renal cell carcinoma (RCC) is one of the most drug-resistant malignancies, and an effective therapy is lacking for metastatic RCC. Anisomycin is known to inhibit protein synthesis and induce ribotoxic stress. The aim of this study was to explore whether anisomycin enhances the cytotoxic effects of mapatumumab, a human agonistic monoclonal antibody specific for death receptor 4 (DR4), in human RCC cells.