Background: Primary type 2 hyperoxaluria is a very rare genetic disorder, where in the progression to renal failure was assumed to be insidious and not very common. PH2 is due to deficient glyoxylate reductase/hydroxypyruvate reductase (GRHPR), which was thought to have extra-hepatic production also. The progression to renal failure in these patient subgroups is well documented in the Literature and the role of SLK (simultaneous liver and kidney transplantation) has not been clearly established.
View Article and Find Full Text PDFAims And Background: To describe the prevalence of IgA deposits (IgAD) in renal allografts in a cohort of renal transplant recipients and to analyze their management strategies and histopathology. To assess graft function and proteinuria after 1 year of follow-up.
Materials And Methods: A prospective longitudinal follow-up study was carried out in VPS Lakeshore Hospital and Research Centre, Kochi, Kerala, over a period of 5 years (July 2015 to June 2020).