Publications by authors named "Jin Haiwei"

Background: Dental pulp tissues are rich in pain-related afferent nerve fibers, which originate from primary sensory neurons in the trigeminal ganglion (TG). The mechanisms of central nervous system (CNS) underlying ectopic pain following peripheral inflammation have been reported that the macrophages as inflammatory and immunologic mediators in the TG play an important role in the process of pulpitis and hyperalgesia.

Objective(s): To observe the polarization response and dynamic distribution of macrophages in the TG during the development of dental pulp inflammation.

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To understand the status and dynamics of macrozoobenthos community in Sanmen Bay, we collected benthos samples by Agassiz trawl in November 2015, and February, May and August in 2016. The results showed that: 1) A total of 119 macrozoobenthos species were recorded, with main groups of fishes, crustaceans and molluscs, accounting for 79% of the total number of species. 2) The dominant species of macrozoobenthos throughout the year were , and .

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Objectives: This study aimed to investigate the role of JAK2-STAT3 (Janus kinase 2/signal transducer and activator of transcription 3) in periapical disease caused by Enterococcus faecalis, as well as the correlation between lipoteichoic acid (LTA) in E. faecalis and the activity of the JAK2-STAT3 signaling pathway and osteoclast formation.

Materials And Methods: A rat model of periapical periodontitis induced by E.

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To determine the expression of Bruton's tyrosine kinase (BTK) in refractory periapical periodontitis and analyze the relationship between BTK and bone resorption in refractory periapical periodontitis. The mechanism of bone resorption is also discussed. The OneArray Plus expression microarray was used to screen for genes related to refractory periapical periodontitis.

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Introduction: We wished to examine the effects of the nucleotide-binding oligomerization domain-like receptor protein 3 (NLRP3) inflammasome on periapical periodontitis induced by Enterococcus faecalis and to investigate the molecular mechanisms of lipoteichoic acid (LTA) derived from E. faecalis on the expression of the NLRP3 inflammasome.

Methods: A model of periapical periodontitis by sealing E.

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Objective: This research aims to study the changes in pain threshold and glial cell line-derived neurotrophic factor (GDNF) in a Sprague Dawley (SD) rat model oftrigeminal neuralgia.

Methods: A total of 36 male SD rats were randomly divided into three groups: operative, sham-operative, and control. In the operative group, a chronic constriction injury (CCI) was caused by placing loose chromic gut ligatures around the right infraorbital nerve (ION).

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An integrative technology including the surfactant enhanced sorption and subsequent desorption and biodegradation of phenanthrene in the soil-water system was introduced and tested. For slightly contaminated agricultural soils, cationic-nonionic mixed surfactant-enhanced sorption of organic contaminants onto soils could reduce their transfer to plants, therefore safe-guarding agricultural production. After planting, residual surfactants combined with added nonionic surfactant could also promote the desorption and biodegradation of residual phenanthrene, thus providing a cost-effective pollution remediation technology.

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A total of 453 Trachurus japonicus specimens with a fork length (FL) of 46-250 mm were sampled in the seasonal light seine net surveys in the East China Sea in May, August, and November, 2008 and in February, 2009. The stomach contents of the specimens were analyzed, and the seasonal and ontogenetic variations in the feeding habits of the T. japonicus were examined by the Kruskal-Wallis test, chi-squared test, and cluster analysis.

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Immunohistochemistry for activated caspase-3 and terminal deoxynucleotidyl transferease-mediated dUTP-biotin nick end labeling (TUNEL) was performed on the trigeminal ganglion after infraorbital nerve transection in newborn rats. The injury induced caspase-3-immunoreactivity and DNA fragmentation in neuronal cell bodies in the maxillary division of the ganglion ipsilateral to the injury. Starting at 16 h post-injury the immunoreactive and TUNEL-positive neurons increased and reached the peak at 24 h (7.

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