Impact of Volume Management on Volume Overload and Rehospitalization in CAPD Patients.

Heart failure due to volume overload is a major reason for rehospitalization in continuous ambulatory peritoneal dialysis patients. Strict volume control provides better cardiac functions and blood pressure in this population. Volume management, which is a volume control strategy, may decrease volume overload and related complications.

Development and Testing of Self-Management Scale for PD Patients.

Objectives: To develop and evaluate the self-management scale for peritoneal dialysis (PD) patients.

Methods: The item pool was formulated based on literature reviews and in-depth interviews. An initial scale containing five factors and 44 items was constructed through two rounds of Delphi expert consultation and a preliminary test.

Establishment of a Weight Management Scale for Patients with Congestive Heart Failure.

Purpose: The aim of this study was to develop a rating scale for the weight management of patients with congestive heart failure (CHF).

Methods: The original pool of items was created through in-depth interviews and a literature review. Scale validity was analyzed based on face validity, content validity, and structure validity.

Reduction of heart failure rehospitalization using a weight management education intervention.

Background: Previous investigators have demonstrated that patient adherence to optimal weight monitoring resulted in fewer heart failure (HF)-related rehospitalizations.

Objective: The aim of this study was to determine whether a weight management (WM) intervention can improve patients' WM ability and cardiac function and reduce HF-related rehospitalizations.

Methods: Heart failure patients were randomly assigned to an intervention group (n = 32) or a control group (n = 34).

Recruitment of phosphoinositide 3-kinase defines a positive contribution of tyrosine kinase signaling to E-cadherin function.

Classical cadherin adhesion molecules can function as adhesion-activated cell-signaling receptors. One key target for cadherin signaling is the lipid kinase phosphoinositide (PI) 3-kinase, which is recruited to cell-cell contacts and activated by E-cadherin. In this study, we sought to identify upstream factors necessary for E-cadherin to activate PI 3-kinase signaling.