40Ni-Added Poly(polyoxometalate) Assembled by {NiGeW} and {Ni(GeW)} Units: Structure, Magnetic, and Heterogeneous Catalysis Properties.

A novel 40Ni-added germanotungstate, CsKNaH{[Ni(OH)(HO)(B-α-GeWO)][Ni(μ-O)(μ-OH) (μ-OH)(HO)BO(OH)(B-α-GeWO)]}·84HO (), was made by the reaction of the trivacant [A-α-GeWO] ({GeW}) precursor with Ni cations and BO, and systematically investigated by Fourier-transform infrared spectroscopy, elemental analysis, thermogravimetric analysis, and powder X-ray diffraction. Single crystal X-ray analysis indicates that the polyoxoanion of is a novel octamer constructed by {NiGeW} and {Ni(GeW)} structural building units via Ni-O═W linkages. The magnetic behavior shows the existence of overall ferromagnetic interactions among the Ni centers in compound .

Corrigendum: The Depression Anxiety Stress Scale-21 in Chinese Hospital Workers: Reliability, Latent Structure, and Measurement Invariance Across Genders.

[This corrects the article DOI: 10.3389/fpsyg.2020.

Corrigendum: The Depression Anxiety Stress Scale-21 in Chinese Hospital Workers: Reliability, Latent Structure, and Measurement Invariance Across Genders.

[This corrects the article DOI: 10.3389/fpsyg.2020.

The Depression Anxiety Stress Scale-21 in Chinese Hospital Workers: Reliability, Latent Structure, and Measurement Invariance Across Genders.

The Depression Anxiety Stress Scale-21 (DASS-21) is an instrument in the assessment of mental health status. The current study recruited 1,532 Chinese hospital workers [74.4% female; mean age = 31.

Serum netrin-1 serves as a prognostic biomarker of aneurysmal subarachnoid hemorrhage.

Background: Netrin-1 exhibits anti-inflammatory properties. Netrin-1 could alleviate brain injury of subarachnoid hemorrhage (SAH) rat. This study was designed to discern the utility of serum netrin-1 as a biomarker for assessing the severity and prognosis of patients with aneurysmal SAH.

The NLRP3 Inflammasome: An Important Driver of Neuroinflammation in Hemorrhagic Stroke.

Hemorrhagic stroke is a devastating clinical event with no effective medical treatment. Neuroinflammation, which follows a hemorrhagic stroke, is an important element that involves both acute brain injury and subsequent brain rehabilitation. Therefore, delineating the key inflammatory mediators and deciphering their pathophysiological roles in hemorrhagic strokes is of great importance in the development of novel therapeutic targets for this disease.