Feeding Habits in the Cultural Domains of Child Care: Elements for Health Promotion.

Family eating behavior is determined by the meaning that the caretaker gives to food and the act of eating in the domestic environment, as well as the beliefs and perceptions around those concepts. Identify the place that nutrition has within the dimensions of child care, the specific weight that the caregiver gives to it within the range of actions deployed and if there are differences when the child exhibits neurodevelopmental disorders, as a contribution to the design of interventions in health promotion. Qualitative, exploratory, two-stage study, with the approach of cognitive anthropology; proposal sampling of maximum differences, 121 informants participated in three groups, caregivers of: (1) healthy children, (2) children who had been hospitalized between 3 and 6 months prior to the time of the interview, and (3) children with a diagnosis of permanent neurological injury and that express some type of neurodevelopmental disorder.

Differential Methylation in Promoter Regions of the Genes and , Involved in the Regulation, and Bioavailability of Cortisol in Leukocytes of Women With Preeclampsia.

Hypertensive disorders are of interest in obstetrics and gynecology because they are the second place among causes of maternal mortality and a source of complications in the short, mid, and long term. Even if the pathophysiological process behind preeclampsia (PE) is still unknown, stress factors have been revealed to play an important role in the genesis of this pathologic process. A case-control study was designed with the purpose of determining if there is a differential methylation in , and genes, related to signaling of the hypothalamic-pituitary-adrenal axis, and its regulation on early-onset PE (EOPE).

Haplotype analysis of TGF-β1 gene in a preeclamptic population of northern Mexico.

Objective: To determine the frequencies of -800G/A (rs1800468), -509C/T (rs1800469) and 869T/C (rs1800470) polymorphisms and their haplotypes in the TGF-β1 gene and their association with preeclampsia in a population of northern México.

Design And Methods: This case-control study involved 175 preeclamptic and 253 normoevolutive pregnant women. The polymorphisms were genotyped by real time PCR.

Genesis of preeclampsia: an epidemiological approach.

THERE ARE ANALYZED SOME OF THE MAIN ASPECTS RELATED TO THE CAUSALITY OF PREECLAMPSIA, PRIVILEGING TWO TYPES OF MODELS: the clinic model and the epidemiologic model, first one represented by the hypothesis of the reduced placental perfusion and the second one considering the epidemiologic findings related to the high levels of psychosocial stress and its association with preeclampsia. It is reasoned out the relevance of raising the causality of the disease from an interdisciplinary perspective, integrating the valuable information generated from both types, clinical and epidemiologic, and finally a tentative explanatory model of preeclampsia is proposed, the subclinical and sociocultural aspects that predispose and trigger the disease are emphasized making aspects to stand out: the importance of reduced placental perfusion as an indicator of individual risk, and the high levels of physiological stress, as a result of the unfavorable conditions of the psychosocial surroundings (indicator of population risk) of the pregnant women.

[The biopsychosocial dimension of preeclampsia: a conceptual-empirical approach].

From the historical point of view, the toxemia of pregnancy have represented one of the most important clinical and epidemiological investigations related to the process of human reproduction. Remains and their significance is due largely to the close relationship that keeps the disease with the figures for maternal mortality, not only in Mexico but throughout the world. Proposes the addition of a guideline to establish a general explanatory principle to link causally the socioepidemiological and psychosocial characteristics of population groups susceptible to the processes clinical, pathophysiological, biochemical and molecular characteristics of disease.