Publications by authors named "Jaime E Gonzalez"

Patients with ventricular noncompaction are susceptible to developing ventricular tachycardia. Commonly, the origin of ventricular tachycardia is endocardial; however, epicardial origins and scar cannot be excluded and should be considered when poor endocardial mapping is present. Other cardiomyopathies, such as arrhythmogenic right ventricular cardiomyopathy, can coexist with ventricular noncompaction and should be excluded in these patients.

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Article Synopsis
  • Assessing the noncoronary cusp and aortomitral continuity is crucial when detecting early atrial activation linked to atrial tachycardias near the His bundle region, especially if P-wave morphology suggests a left atrial origin.!* -
  • For patients with congenital heart anomalies, it’s important to explore different catheter positioning methods, such as using retrograde access for left atrial tachycardias and placing intracardiac echocardiography in the azygous vein to better visualize heart structures.!* -
  • Utilizing remote magnetic navigation may be a beneficial alternative approach if it’s available for managing these atrial conditions.!*
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Atrioventricular reciprocating tachycardia is a common cause of undifferentiated supraventricular tachycardia. In patients with manifest or concealed accessory pathways, it is imperative to assess for the presence of other accessory pathways. Multiple accessory pathways are present in 4% to 10% of patients and are more common in patients with structural heart disease.

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This case-based review discusses the benefits of cardiac resynchronization therapy (CRT) and whether defibrillation function is necessary in CRT responders. An evaluation of the literature and evidence to date is discussed. Recommendations based on these data, expert opinion, and recently published appropriate use criteria are given.

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Amiodarone is an effective treatment for atrial and ventricular arrhythmias, but its use is limited by a toxic adverse-effect profile. Although dronedarone has been touted as an antiarrhythmic agent devoid of both solid organ toxicity and proarrhythmic properties, its potential for prolonging ventricular repolarization may augment triggered ectopy. We describe a 66-year-old man who began dronedarone 400 mg twice/day for new-onset paroxysmal atrial fibrillation; he had no left ventricular dysfunction or clinical heart failure.

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