Publications by authors named "J Mogan"

Gait recognition, the task of identifying an individual based on their unique walking style, can be difficult because walking styles can be influenced by external factors such as clothing, viewing angle, and carrying conditions. To address these challenges, this paper proposes a multi-model gait recognition system that integrates Convolutional Neural Networks (CNNs) and Vision Transformer. The first step in the process is to obtain a gait energy image, which is achieved by applying an averaging technique to a gait cycle.

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Additive manufacturing (AM) highlights developing complex and efficient parts for various uses. Fused deposition modelling (FDM) is the most frequent fabrication procedure used to make polymer products. Although it is widely used, due to its low characteristics, such as weak mechanical properties and poor surface, the types of polymer material that may be produced are limited, affecting the structural applications of FDM.

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Identifying an individual based on their physical/behavioral characteristics is known as biometric recognition. Gait is one of the most reliable biometrics due to its advantages, such as being perceivable at a long distance and difficult to replicate. The existing works mostly leverage Convolutional Neural Networks for gait recognition.

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Identifying people's identity by using behavioral biometrics has attracted many researchers' attention in the biometrics industry. Gait is a behavioral trait, whereby an individual is identified based on their walking style. Over the years, gait recognition has been performed by using handcrafted approaches.

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MPYS (also known as STING, MITA, and TMEM173) is a type I IFN stimulator that is essential for host defense against DNA virus infection and appears important in defense against certain bacteria. The in vivo significance and mechanisms by which MPYS mediates host defense against nonviral pathogens are unknown. Using an MPYS-deficient mouse (Tmem173()), we determined that, distinct from the IFNAR(-/-) mice, MPYS deficiency leads to increased bacterial burden in the liver upon Listeria monocytogenes infection.

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