The obesogenic effects of Bisphenol A and its analogues are differentially regulated via PPARγ transactivation in mouse 3T3-L1 cells.

Exposure to environmental pollutants with obesogenic activity is being recognised as one of the contributing factors to the obesity epidemic. Bisphenol A (BPA) has been shown to stimulate adipogenesis in both human and mouse preadipocytes, to increase body weight and affect lipid metabolism in animal and epidemiological studies. Regulatory action and public concern has prompted industry to replace BPA with other structurally similar analogues that may have similar effects.

Genome Sequence of Phage .

Phage JanetJ was isolated on and has siphovirus morphology. JanetJ's genome consists of 36,986 base pairs, encoding 52 putative protein-coding genes. JanetJ adds to the small number of previously isolated cluster FO phages, none of which encode identifiable immunity repressor or integrase functions, with the exception of phage Maja.

Tetra methyl bisphenol F: another potential obesogen.

Background/objectives: Obesity and its associated metabolic diseases are increasing globally. Sedentary lifestyle, high caloric diet, and genetic predisposition are known to contribute to the onset of obesity. It is increasingly recognized that exposure to environmental chemicals such as Bisphenol A (BPA) may also play a significant role.

Low dose antibiotic ingestion potentiates systemic and microbiome changes induced by silver nanoparticles.

Changes in the mammalian gut microbiome are linked to the impairment of immunological function and numerous other pathologies. Antimicrobial silver nanoparticles (AgNPs) are incorporated into numerous consumer products (e.g.

The Complete Genome Sequence of Bacillus thuringiensis Strain ATCC 13367 Features a -Containing Chromosome.

We report the high-quality closed genome sequence of Bacillus thuringiensis ATCC 13367, which is used as a bioinsecticide. The genome features a 328-kbp plasmid and a 5.8-Mbp chromosome.