[Changes in the nitrite level of the blood in patients in the acute period of a subarachnoid hemorrhage of aneurysmatic etiology].

Subarachnoidal hemorrhage leads to impaired endothelium-dependent relaxation by inhibiting the synthesis of major vascular relaxation agent nitric oxide (NO). Nitrite, a closest product of the oxidation of NO, is responsible for the synthesis of the latter. The Griss reaction was used for daily measurement of blood nitrite levels in 32 patients with acute subarachnoidal hemorrhage of aneurysmal etiology.