[Clinical and electrophysiologic variants of false ventricular tachycardia (supraventricular tachycardia with wide QRS complex)].

Electrophysiological abnormalities of the heart conduction system and their role in development of different forms of supraventricular tachycardias, are discussed. Electrophysiological mechanisms and diagnostic criteria of the supraventricular tachycardias with wide QRS complex, are described.

[Efficiency of antiarrhythmic therapy and resistance to it in patients with malignant ventricular tachyarrhythmias: formulation of prognosis].

Aim: To study feasibility of predicting results of drug antiarrhythmic therapy in patients with malignant ventricular arrhythmias (MVA) basing on the results of clinical and device examinations.

Materials And Methods: 136 patients with documented MVA entered the study. 100 patients were in the retrospective analysis group, 36 patients comprised the study group.

[Permanent repetitive nonparoxysmal ventricular tachycardia. II. The results of using anti-arrhythmia preparations and the characteristics of its long-term course].

A total of 20 patients with repetitive nonparoxysmal ventricular tachycardia (RNVT) underwent Holter ECG monitoring to test efficacy of successive oral antiarrhythmic drugs. The response was recorded in 50% of the patients. Although the effect of the drugs persisted for a few months only, RNVT ran without severe symptoms, in contrast to paroxysmal ventricular tachycardia.

[Permanent repetitive nonparoxysmal ventricular tachycardia. I. The electrocardiographic manifestations and possible mechanisms of its occurrence].

A total of 20 patients (16 males and 4 females) with repetitive nonparoxysmal ventricular tachycardia (RNVT) underwent Holter monitoring of ECG, bicycle exercise test, intracardiac electrophysiological measurements. The findings evidence for a principal difference of this tachycardia form from paroxysmal ventricular tachycardia by trigger mechanism. All the tests performed showed rhythm-dependence of RNVT, tachycardia cannot be initiated by electrocardiostimulation, but can be stopped after intravenous introduction of finoptin.