[HMGB1 neutralizing antibody attenuates hemorrhagic shock-induced cardiac injury and modulates apoptotic gene expression in murine heart].

Objective: To explore the possible protective effects and mechanisms of neutralizing antibody of HMGB1 on hemorrhagic shock-induced cardiac injury in mice.

Methods: The KM mice of hemorrhagic shock model were divided into sham (sham), control+IgG, hemorrhagic shock (HS) and HMGB1 neutralizing antibody treatment (HS+α-HMGB1) groups (n = 8 each). After modeling, the animals were anesthetized and blood samples collected.

A new concept of desulfurization: the electrochemically driven and green conversion of SO2 to NaHSO4 in aqueous solution.

A new concept of desulfurization was developed by designing a series of electrochemical reactions to drive an SO2 absorption-and-conversion process in aqueous solution, hence the SO2 in gas was eventually converted to a valuable chemical of NaHSO4. A model experiment of chemically substantiating this concept includes two steps: (I) absorption of SO2 gas by aqueous solution and oxidation of the absorbed SO2 to SO4(2-) by air and (II) transformation of the SO4(2-) to NaHSO4. The experiment demonstrated that in Step I, the cathodic reduction of 02 from ambient air scavenged the H+ released due to the SO2 absorption and its further oxidation, which thereby were accelerated.

A novel electro-fenton process for water treatment: reaction-controlled pH adjustment and performance assessment.

A novel electro-Fenton (E-Fenton) process was developed, in which the desired pH for an effective E-Fenton reaction and for a neutral treated media could be obtained by utilizing the reaction-released H+ and OH- in stead of chemical addition. In the laboratory-scale process using three chambers, the substrate solution pH > 4.0 was designed to be adjusted in situ through three sequencing steps: (I) pH reduction, (II) pH keeping for the effective E-Fenton reaction, and (III) pH recovery to neutral while the E-Fenton reaction continued.