Background: Office workers are consistently exposed to blue light, mainly from sunlight and digital device. Recent studies report that blue light has various harmful effects, including cellular changes via reactive oxygen species. Studies on blue light-induced skin changes have only been conducted in vitro and have not been clinically confirmed.
View Article and Find Full Text PDFJ Cosmet Dermatol
September 2020
Background: Blue light from electronic devices enriched with a peak at 456 nm affects circadian rhythm and antioxidant balance of skin, necessitating the study of photoprotection against the 456-nm blue light.
Aims: This study aims to report that blue light (456 nm) can cause skin pigmentation and proposes a new clinical evaluation method for blue light (456 nm) protection based on the skin pigmentation level.
Patients/methods: We developed a clinical device (ABC device ) that emits blue light (peak = 456 nm).
Lanthanide-doped upconverting nanoparticles (UCNPs, NaYF4:Yb(3+),Er(3+)) are well known for emitting visible photons upon absorption of two or more near-infrared (NIR) photons through energy transfer from the sensitizer (Yb(3+)) to the activator (Er(3+)). Of the visible emission bands (two green and one red band), it has been suggested that the red emission results from two competing upconversion pathways where the non-radiative relaxation occurs after the second energy transfer (pathway A, (4)I15/2 → (4)I11/2 → (4)F7/2 → (2)H11/2 → (4)S3/2 → (4)F9/2 → (4)I15/2) or between the first and the second energy transfer (pathway B, (4)I15/2 → (4)I11/2 → (4)I13/2 → (4)F9/2 → (4)I15/2). However, there has been no clear evidence or thorough analysis of the partitioning between the two pathways.
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