Preparation and Characterization of Thermoelectric PEDOT/Te Nanorod Array Composite Films.

In this study, we prepared Te nanorod arrays via a galvanic displacement reaction (GDR) on a Si wafer, and their composite with poly(3,4-ethylenedioxythiophene) (PEDOT) were successfully synthesized by electrochemical polymerization with lithium perchlorate (LiClO) as a counter ion. The thermoelectric performance of the composite film was optimized by adjusting the polymerization time. As a result, a maximum power factor (PF) of 235 µW/mK was obtained from a PEDOT/Te composite film electrochemically polymerized for 15 s at room temperature, which was 11.

Facial Fabrication and Characterization of Novel Ag/AgCl Chloride Ion Sensor Based on Gel-Type Electrolyte.

In this study, a novel chloride ion (Cl) sensor based on Ag wire coated with an AgCl layer was fabricated using a gel-type internal electrolyte and a diatomite ceramic membrane, which played an important role in preventing electrolyte leakage from the ion-selective electrode. The sensing performance, including reversibility, response, recovery time, low detection limit, and the long-term stability, was systemically investigated in electrolytes with different Cl contents. The as-fabricated Cl sensor could detect Cl from 1 to 500 mM KCl solution with good linearity.

Senescence-related functional nuclear barrier by down-regulation of nucleo-cytoplasmic trafficking gene expression.

One of the characteristic natures of senescent cells is the hypo- or irresponsiveness not only to growth factors but also to apoptotic stress. In the present study, we confirmed the inhibition of nuclear translocation of activated p-ERK1/2 and NF-kB p50 in response to growth stimuli or LPS in the senescent human diploid fibroblasts. In order to elucidate the underlying mechanism for the senescence-associated hypo-responsiveness, we carried out the comparison study for gene expression profiles through microarray analysis.

Perilla leaf, Perilla frutescens, induces apoptosis and G1 phase arrest in human leukemia HL-60 cells through the combinations of death receptor-mediated, mitochondrial, and endoplasmic reticulum stress-induced pathways.

Since it has been reported that Perilla leaves (Perilla frutescens) have antimutagenic, antioxidant, and anti-inflammatory properties, we hypothesized that Perilla leaves may have a potential anticancer activity. Therefore, we examined the possibility that cancer cell growth is reduced by treatment with a Perilla leaf ethanol extract (PLE) using human leukemia HL-60 cells and then investigated the mechanism of the growth inhibition. We found that PLE treatment suppressed cell viability in a dose-dependent manner.