The Rice Locus Is a Bidirectional Target of the African pv. Major Transcription Activator-like Effector TalC but Does Not Contribute to Disease Susceptibility.

pv. () strains that cause bacterial leaf blight (BLB) limit rice () production and require breeding more resistant varieties. Transcription activator-like effectors (TALEs) activate transcription to promote leaf colonization by binding to specific plant host DNA sequences termed effector binding elements (EBEs).

A Pathovar of Infecting Wild Grasses Provides Insight Into the Evolution of Pathogenicity in Rice Agroecosystems.

() are globally important rice pathogens. Virulent lineages from Africa and Asia and less virulent strains from the United States have been well characterized. pv.

Functional and Genome Sequence-Driven Characterization of Effector Gene Repertoires Reveals Novel Variants With Altered Specificities in Closely Related Malian pv. Strains.

Rice bacterial leaf blight (BLB) is caused by pv. () which injects Transcription Activator-Like Effectors (TALEs) into the host cell to modulate the expression of target disease susceptibility genes. major-virulence TALEs universally target susceptibility genes of the SWEET sugar transporter family.

Targeted promoter editing for rice resistance to Xanthomonas oryzae pv. oryzae reveals differential activities for SWEET14-inducing TAL effectors.

As a key virulence strategy to cause bacterial leaf blight, Xanthomonas oryzae pv. oryzae (Xoo) injects into the plant cell DNA-binding proteins called transcription activator-like effectors (TALEs) that bind to effector-binding elements (EBEs) in a sequence-specific manner, resulting in host gene induction. TALEs AvrXa7, PthXo3, TalC and Tal5, found in geographically distant Xoo strains, all target OsSWEET14, thus considered as a pivotal TALE target acting as major susceptibility factor during rice-Xoo interactions.