Publications by authors named "H M S Farah"
Background: Non-malignant tumors of the CNS contribute substantially to the morbidity and mortality from CNS tumors. It is critical to understand the epidemiology of non-malignant CNS tumors separately from CNS malignancies to inform resource allocation and policy since treatment and prognosis can differ. High quality international data on non-malignant CNS tumor burden are needed to accomplish this goal.
View Article and Find Full Text PDFBackground/objectives: In contrast to traditional biometric modalities, such as facial recognition, fingerprints, and iris scans or even DNA, the research orientation towards chest X-ray recognition has been spurred by its remarkable recognition rates. Capturing the intricate anatomical nuances of an individual's skeletal structure, the ribcage of the chest, lungs, and heart, chest X-rays have emerged as a focal point for identification and verification, especially in the forensic field, even in scenarios where the human body damaged or disfigured. Discriminative feature embedding is essential for large-scale image verification, especially in applying chest X-ray radiographs for identity identification and verification.
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- Managing hemodynamic instability in critically ill patients involves a balanced approach to treat underlying cardiovascular issues while addressing their systemic effects, requiring constant monitoring and adaptability in treatment strategies.
- A study conducted with 300 nurses in Palestine utilized a questionnaire to assess their knowledge, attitudes, and practices regarding hemodynamic instability in cardiovascular disease patients.
- Results indicated that while nurses had low knowledge levels about hemodynamic instability (mean score 2.7/7), they maintained positive attitudes toward their practice (mean score 3.8/5), with some variation based on gender and education level.
Article Synopsis
- Alpha-1 antitrypsin (AAT) deficiency is a genetic disorder mainly caused by a specific mutation, leading to severe lung and liver issues, which increase mortality rates.
- The E342K mutation alters the AAT protein, causing its accumulation in the liver and reducing its secretion, which results in lung damage due to unopposed protease activity.
- New treatments, particularly genomic medicines like RNA editing, show promise for correcting the mutation and restoring normal AAT levels for better management of both lung and liver conditions.
Article Synopsis
- This study looked at how early treatment helps babies with spinal muscular atrophy (SMA), a condition that affects muscles.
- Researchers checked the health of 66 babies treated within the first 6 weeks of life and found good results, like all babies being able to sit up on their own.
- However, some babies with a certain genetic marker (two SMN2 copies) had a harder time walking compared to those with more copies, showing that more studies are needed to improve their care.