We previously reported that cubital venous pressure (Pv) tended to increase initially, but this was followed by a drop in a dose-dependent response after intravenous lidocaine administration in subjects with various diseases. In this study we examined whether Pv responses after small-dose intravenous lidocaine administrations are related to the stimulating effect of lidocaine on vascular smooth muscle (VSM). In 5 subjects free of cardiovascular disease, Pv increased slightly with decreased pulsations after a 10 mg dose (p<0.
View Article and Find Full Text PDFThe aim of the study is to establish the validity of the use of the electrical impedance technique for estimating local oedema in extremities through comparison with computed-tomography (CT) analysis. Eight healthy women (group 1) and nine women with unilateral leg oedema following chronic lymphatic obstruction (group 2) are examined sequentially utilising both methods. 'Equivalent resistivities' of extra- and intracellular fluid (RE and RI) are measured in the leg's upper portion with a multi-frequency impedance meter, and seven CT slices are taken in the same leg segments.
View Article and Find Full Text PDFRupture of a chronic postinfarction aneurysm of the left ventricle is rare and usually fatal. A 63-year-old man with rupture of a true left ventricular aneurysm 67 days after myocardial infarction was treated successfully with resection of the aneurysm. The postoperative course was uneventful.
View Article and Find Full Text PDFA 50-year-old woman was admitted to the intensive care unit 40 min after the onset of severe chest and back pain with consciousness loss. Emergency computed tomography and aortography demonstrated an acute type A dissection without opacification of the false lumen. The patient was initially treated with antihypertensive drugs.
View Article and Find Full Text PDFA case of histologically proven dilated cardiomyopathy and a case of clinically diagnosed cardiomyopathy (cardiac amyloidosis was strongly suspected but was not confirmed) were examined with 99mTc(V)-dimercaptosuccinic acid (DMSA). 99mTc(V)-DMSA accumulation in the damaged myocardium was clearly demonstrated. These results suggested the possibility that 99mTc(V)-DMSA could be used as a positive agent for cardiomyopathy.
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