Extreme thermophilic microbial inoculation for reducing NH and NO emissions in hyperthermophilic aerobic composting of refinery waste activated sludge.

Ammonia (NH) and nitrous oxide (NO) release are the main causes of nitrogen loss during aerobic composting. In this study, hyperthermophilic aerobic composting of refinery waste activated sludge (RWAS) was performed by adding extreme thermophilic bacteria, and the effects of inoculation on NH and NO emissions were systematically studied. The results revealed that inoculation achieved hyperthermophilic aerobic composting (T group), increased maturity, and reduced NH and NO emissions by 32.

Regulates Muscle Growth and Development by Targeting .

Non-coding genes, such as microRNA and lncRNA, which have been widely studied, play an important role in the regulatory network of skeletal muscle development. However, the functions and mechanisms of most non-coding RNAs in skeletal muscle regulatory networks are unclear. This study investigated the function and mechanism of in muscle growth and development.

The correlation between calcaneocuboid joint involvement and sustentacular fragment displacement among male patients with displaced intra-articular calcaneal fractures.

Sustentacular fragment displacement is not uncommon in calcaneal fractures. The factors that suggest a possible displacement of sustentacular fragment are still unclear. Displaced intra-articular calcaneal fractures with calcaneocuboid joint involvement are the most common type of calcaneal fracture.

Effects and interaction of humidex and air pollution on influenza: A national analysis of 319 cities in mainland China.

Influenza imposes a significant global health burden. This study investigates the effects of humidex and air pollution on influenza and their interactions, using multi-city surveillance data in China. Daily data on reported influenza cases, meteorological factors and air pollution from 319 cities in mainland China over the study period of 2014-2019 were collected.

Gastrodin inhibits reactive astrocyte-mediated inflammation in hypoxic-ischemic brain damage through S100B/RAGE-Smad3 signaling.

Activated astrocytes and their associated inflammatory responses play critical roles in the pathogenesis of hypoxic-ischemic brain damage (HIBD). Gastrodin (GAS), an anti-inflammatory herbal agent, is known to suppress microglial activation. Here, we investigate whether it exerts a similar effect on activated astrocytes and whether it acts through S100B/RAGE-Smad3 signaling.