Tachycardia-induced cardiomyopathy (TIC) is a reversible subtype of dilated cardiomyopathy (DCM) resulting from sustained supraventricular or ventricular tachycardia and diagnosed by the normalization of left ventricular ejection fraction (LVEF) after stable sinus rhythm restoration. The aim of this study was to determine the contribution of cardiac magnetic resonance (CMR) to the differential diagnosis of TIC and DCM with persistent atrial arrythmias in patients hospitalized for the first time with heart failure (HF) with reduced LVEF of nonischemic origin. A total of 29 patients (age: 58.
View Article and Find Full Text PDFThe tachycardia-induced cardiomyopathy is a rare case of reversible heart failure and left ventricle disfunction. The diagnostic approach and treatment strategy are described in this article. Also the clinical case of heart failure compensation in the patient with left ventricle dilatation and atrial flutter after the reverse to sinus rhythm is after catheter ablation presented.
View Article and Find Full Text PDFA 56-year-old male with paroxysmal atrial fibrillation refractory to class IC and class III antiarrhythmic drugs was admitted to our hospital for radiofrequency catheter ablation of atrial fibrillation. During preoperative examination situs inversus totalis was revealed. Pulmonary vein (PV) isolation was successfully performed with atrial fibrillation termination and elimination of all PV potentials.
View Article and Find Full Text PDFBackground: Recent studies have suggested that para-Hisian atrial tachycardia (AT) can be successfully ablated from the right atrium (RA), left atrium (LA), or noncoronary cusp (NCC) in the aorta; however, the best approach remains unclear.
Objective: This study aimed to compare different approaches to radiofrequency catheter ablation (RFCA) of para-Hisian AT.
Methods: We retrospectively analyzed 68 consecutive patients (49[72%] women; mean age 61 ± 13 years) treated with RFCA for this type of AT.
Purpose: To assess characteristics of involvement of coronary vascular bed in patients with ischemic heart disease and cardioverter defibrillator (CD) implanted for primary prevention of sudden cardiac death (SCD) with and without history of appropriate implantable (I) CD therapies.
Material And Methods: We examined 64 survivors of myocardial infarction (MI) (60 men, 4 women) with CD implanted for primary prevention of SCD. Mean duration of follow up after CD implantation was 19.
The article describes the data of efficiency of catheter ablation of substrate of ventricular arrhythmias, performed in patients with ischemic heart disease and implanted cardioverter- defibrillator (ICD) in order to decrease the number of ICD therapy. An assessment of the follow-up study of 32 patients was made, the catheter ablations were performed in 10 of them. The statistically reliable number of ICD therapy and at the same time both "shock" and antitachycardia pacing decreased in the group of catheter ablation.
View Article and Find Full Text PDFDynamics of blood pressure (BP) and heart rate (HR) was traced by automatic monitoring every 30 min uninterruptedly along several months in a patient suffering from combined atrial fibrillation and heart failure during the development of disease and its therapeutic and surgical treatment (pacemaker implanting and atrioventricular ablation). Analyses of spectral components as well as signal's shape revealed instabilities in circadian and semicircadian parameters. A new approach for signal's form description without using cosine approximation is suggested.
View Article and Find Full Text PDFNew data about the development of placental circulation and the features of its regulation in normal and complicate pregnancy are considered in present review. Biochemical mechanisms leading to the dilatation of umbilical-placental circulation as well as the relaxation and contraction of placental vessels smooth muscles are discussed. It is shown that the disturbance of the balance between dilation and constriction factors being the results of injury of endothelial cells causes the spasm of uterine vessels, "delimiting" of maternal circulation from fetal one and proceeds a clinical manifestations of placental insufficiency.
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