* This study found that the analgesic effect of oxytocin is linked to the vasopressin-1a (V1a) receptor, as its effects were blocked by a specific antagonist for this receptor but not by an oxytocin receptor antagonist.
* The research revealed that V1a receptors are heavily expressed in pain-related neurons and that oxytocin reduces calcium activity induced by capsaicin (a pain trigger) and increases potassium flow in these neurons, contributing to its pain-relieving effects.