Murine acquired immunodeficiency syndrome (MAIDS) is caused by exposure to murine leukemia virus and serves as a model to study human AIDS. In MAIDS-susceptible C57BL/6 mice, virus exposure leads to progressive immune deficiency, while resistant strains such as BALB/c recover from infection and develop protective immunity. The goal of this study was to identify early gene expression patterns that may be important in establishing this strain-specific differential response.
View Article and Find Full Text PDFThe inability to consistently achieve a passive fit with screw-retained implant prostheses is well documented in the literature. Cement-retained implant frameworks have been advocated for implant-retained fixed partial dentures. However, cement retention has not been routinely advocated for fixed-detachable hybrid prostheses.
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