Publications by authors named "G TEICHMANN"

Article Synopsis
  • Cervical squamous cell carcinomas (CSCCs) linked to uterine prolapse show limited prior research, prompting a detailed study of their morphologic and molecular traits, including the identification of a new precursor lesion termed HPV-independent cervical intraepithelial neoplasia (CIN).
  • The study involved four patients, primarily older women with advanced disease and TP53 mutations, none showing the common HPV DNA typically associated with cervical cancer.
  • The precursor lesions displayed characteristics akin to differentiated vulvar intraepithelial lesions, suggesting a unique tumorigenetic pathway that differs from typical HPV-related processes.
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Infiltration of surface water constitutes an important pillar in artificial groundwater recharge. However, insufficient transformation of organic carbon and nutrients, as well as clogging of sediments often cause major problems. The attenuation efficiency of dissolved organic carbon (DOC), nutrients and pathogens versus the risk of bioclogging for intermittent recharge were studied in an infiltration basin covered with different kinds of macrovegetation.

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Background: Radical hysterectomy based on empirical surgical anatomy to achieve a wide tumour resection is currently applied to treat early cervical cancer. Total mesometrial resection (TMMR) removes the embryologically defined uterovaginal (Müllerian) compartment except its distal part. Non-Müllerian paracervical and paravaginal tissues may remain in situ despite their possible close proximity to the tumour.

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Aquatic fulvic acid (FA) and humic acid (HA) were characterized by an aqueous high-performance size-exclusion chromatography (HPSEC) using a hydrophilic polymeric stationary phase and an aqueous eluent at neutral pH and low-ionic strength (5 mM Na2HPO4; final ionic strength, 13 mM). Employed HPSEC showed low sensitivity of FA to variations in ionic strength (13 and 100 mM) and contents of organic modifier (0.1 or 40% methanol) in aqueous eluent.

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TIE2 is a vascular endothelial-specific receptor tyrosine kinase essential for the regulation of vascular network formation and remodeling. Previously, we have shown that the 1.2-kb 5' flanking region of the TIE2 promoter is capable of directing beta-galactosidase reporter gene expression specifically into a subset of endothelial cells (ECs) of transgenic mouse embryos.

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