Ethanol-induced changes in lipid peroxidation and glutathione content in rat brain.

The effect of acute and chronic ethanol administration on brain lipid peroxide and glutathione levels was investigated in rats. Acute ethanol administration (5 g/kg, i.p.

Mitochondrial and microsomal lipid peroxidation in rat liver after acute acetaldehyde and ethanol intoxication.

The effects of acetaldehyde and ethanol treatments on hepatic lipid peroxidation are compared in rats after the administration of toxically corresponding doses. Wistar rats were given a single dose of acetaldehyde (0.5 g/kg) or ethanol (5 g/kg) intraperitoneally.

The effect of cholesterol feeding on lipid peroxide, glutathione, glutathione peroxidase and glutathione transferase in the liver of rats.

Liver cholesterol, phospholipid, triglyceride, lipid peroxide and glutathione levels as well as glutathione peroxidase and glutathione transferase activities were determined in rats fed a high-cholesterol (2%, w/w), high-cholic acid (0.5%, w/w) diet for 3 months. Cholesterol feeding caused an increase in hepatic cholesterol and triglyceride levels, but no change was observed in hepatic phospholipid levels.

Erythrocyte lipid peroxidation, glutathione and vitamin E levels in cholesterol-fed rats.

Erythrocyte cholesterol and phospholipid levels, the susceptibility of erythrocytes to lipid peroxidation as well as erythrocyte glutathione and vitamin E levels were determined in rats fed a high cholesterol (2%, w/w) and high cholic acid (0.5%, w/w) diet for 3 months. Cholesterol feeding caused an increase in erythrocyte cholesterol levels, but no change was observed in erythrocyte phospholipid levels.