Determination of Optimum Parameters for Flotation of Galena: Effect of Chain Length and Chain Structure of Xanthates on Flotation Recovery.

The structure of the xanthates' hydrocarbon (C-H) chain is one of the major factors which affect flotation recovery. The effectiveness and the collecting power of xanthates increase with increasing chain length and vary depending on the chain structure: branched and straight chains. In this regard, the influences of length (2-5 carbon) and structure (straight: normal and branched: iso) of xanthate's hydrocarbon chain on flotation recovery of galena were investigated under different experimental conditions: xanthate concentration, conditioning time, air flow rate (AFR), and air bubble size.

Immunohistochemical and histopathological evaluation of 2,4-dichlorophenoxyacetic acid-induced changes in rat kidney cortex.

This study aims to investigate the effect of 2,4-dichlorophenoxyacetic acid (2,4-D) on rat kidney cortex histology. Oral exposure of rats to 2,4-D for 28 days resulted in decreases in body weight gain and kidney weight. Histological examination showed degeneration in renal corpuscles and podocytes; vacuolization in the glomerulus with disintegration of the basal membrane; tissue edema; vacuolization, cystic dilation and invagination of the basal laminae in the tubular structures; dilation and congestion in renal corpuscular vessels and marked decrease in glomerular and stromal fibronectin reaction; suggesting that subacute 2,4-D administration induces dose-dependent histopathological degenerative effects in rat kidney cortex.

[Morphometric variations of the rumen ciliate, Ophryoscolex purkynjei Stein, 1858 (Ciliophora: Entodiniomorphida) from cypriot domestic goats].

The species Ophryoscolex purkynjei and its four formae [O. p. f.

The association between factor XIII Val34Leu polymorphism and early myocardial infarction.

Background: Activated factor XIII (FXIII) cross-links between fibrin monomers, thus increasing the clot stability and resistance to fibrinolysis. Congenital FXIII deficiency causes severe bleeding diathesis. Recently, a common polymorphism of the FXIII A subunit (FXIII Val34Leu) has been identified as a protective factor against both arterial and venous thrombosis.