The peculiarities of the genesis of electrocardiosignal under myocardial ischemia in connection with an increase of its electrical inhomogeneity, as well as the electrophysiological mechanisms of morphological changes of the T wave of the electrocardiogram, including its "symmetrization" have been considered. A systemic approach to the problem has been used, which combines the mathematical, computer, and physiological modeling of the cardiac electrical activity with studies of the electric field of the human heart in terms of biophysical models. A database for the repolarization parameters of experimental electrocardiosignals ("Norm" and "Ischemia" samples) has been formed.
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