Incidence of Brain Vascular Damage in a Population With Parkinson's Disease: Statistical Comparison by Age Subassemblies With Age Homogeneous Control Groups.

We recently published studies on the incidence significance of cerebrovascular disease (CVD) in Parkinson's disease (PD) and some related movement disorders. The CVD was assessed by including lesions morphologically diagnosed by magnetic resonance imaging (MRI) or computed tomography (CT) scan studies that defined ischemic vascular damage with a range from leukoaraiosis to multiple microinfarcts with various distribution, minor stroke, and major stroke and hemorrhagic lesions. These lesions were detected at the beginning of Parkinsonian symptoms or earlier; in any case, all the patients were diagnosed as PD patients according to the international classification and response to therapy.

Vascular Parkinsonism sensitive to Rotigotine therapy is found in aged patients: a clinical case description.

Previous and recent papers have pointed out several discussions about the so called 'Vascular Parkinsonism' (VP), particularly about some distinctive characteristics with respect to the Idiopathic Parkinson Disease (IPD); differences commonly described are  sudden onset of extrapyramidal symptoms after a stroke, main involvement of lower limbs in diffuse brain microinfarcts (lower body parkinsonism), low, short in time or absent response to classic IPD therapy in all kinds of vascular brain pathologies. But few published studies have also emphasized some relationship linking brain morphological signs of vascular damage in elder patients suffering from diagnosed IPD, both 'normally' responding to classic Levodopa therapy or not.

Incidence of vascular brain damage in a population with Parkinson Disease: a clinical statistic study in comparison with a control group of patients afferent to neurological movement disorder outpatients clinic.

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P3 components evoked by unilateral and bilateral somatosensory stimulation: normative data for the study of neglect syndrome.

Latency and amplitude of an "odd-ball" somatosensory P3 component evoked either by unilateral or bilateral somatosensory target stimulation, were measured in 15 healthy right-handed subjects (age range 42-79 years) in order to obtain normative data useful for studying the neglect syndrome. The bilateral stimulation protocol was designed to investigate the tactile extinction phenomenon. P3 waves were recorded from nine electrodes.

Brain excitability and long latency muscular arm responses: non-invasive evaluation in healthy and parkinsonian subjects.

Thirty healthy and 35 volunteers affected by Parkinson's disease (PD) were examined. Long latency responses (LLRs) and short latency somatosensory evoked potentials (SEPs) after median nerve stimulation were respectively recorded from forearm flexor muscles, and from 19 scalp electrodes, during relaxation (condition 1), light and maximal muscle contraction (conditions 2 and 3). Linear interpolation of SEPs was performed to produce isopotential colour maps.