Publications by authors named "F Salmani"

Background And Aims: Mounting evidence have implicated that rs1801131 and rs1801133, located in the Methylenetetrahydrofolate reductase (MTHFR) gene, may emerge as novel biomarkers for coronary artery disease (CAD). The Synergy between Percutaneous Coronary Intervention with Taxus and Cardiac Surgery (SYNTAX) score is also an appropriate predictor for revascularization strategy in patients with complex CAD. The aim of this study is to investigate the correlation between rs1801131 and rs1801133 with the severity of coronary lesions in patients with ST‑Elevation Myocardial Infarction (STEMI) and Non‑ST‑Elevation Myocardial Infarction (NSTEMI) based on the SYNTAX score.

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Acetyl-11-keto-β-boswellic acid is one of the main active components of Boswellia sp. resin with the most potent anti-inflammatory activity. In recent years, herbal therapy has received considerable attention for the treatments of inflammatory and demyelinating diseases such as Multiple sclerosis (MS).

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Background: Prehypertension is one of the common disorders during pregnancy. Reducing salt intake is among the best cost-effective interventions to reduce the risk of hypertension. The present study aimed to explore the effect of an educational intervention based on the Theory of Planned Behavior (TPB) on amount of salt consumed by prehypertensive pregnant women.

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Background: The toxicity of silver nanoparticles (AgNPs) has been proven in the female reproductive system. Thymoquinone (TQ) is a natural antioxidant and bioactive component of .

Objective: We evaluated the efficacy of TQ on ovarian tissue following toxicity induced by AgNPs in female mice.

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Article Synopsis
  • Alzheimer's disease (AD) is influenced by environmental factors like air pollution, but the exact way pollution leads to neurodegeneration is still unclear.
  • The study focused on the role of tau proteins, especially the harmful cis conformation of phosphorylated tau (p-tau), in relation to air pollution exposure in mice.
  • Results indicated that pollution increased the levels of neurotoxic cis p-tau and decreased the Pin1 protein, suggesting a potential link between air pollution and tau-related pathology in AD.
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